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Nat Rev Cardiol [JOURNAL]

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Anticoagulation in patients with rheumatic heart disease and atrial fibrillation: implications for low-resource regions.

Markos S

Nat Rev Cardiol · 2026 Apr · PMID 41360984 · Publisher ↗

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CCTA assessment of atherosclerotic plaque composition to guide coronary artery interventions.

Storozhenko T

Nat Rev Cardiol · 2026 Mar · PMID 41299154 · Publisher ↗

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Redefining heart failure subtypes according to skeletal muscle mass.

Suthahar N

Nat Rev Cardiol · 2026 Feb · PMID 41291290 · Publisher ↗

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Linking cardiovascular disease to cancer progression.

Zambrano A

Nat Rev Cardiol · 2026 Feb · PMID 41291289 · Publisher ↗

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When stents become plaques: in-stent neoatherosclerosis.

Liu J, Bursill C

Nat Rev Cardiol · 2026 Feb · PMID 41291288 · Publisher ↗

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PCSK9 inhibition for primary prevention of cardiovascular events.

Lim GB

Nat Rev Cardiol · 2026 Jan · PMID 41291287 · Publisher ↗

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Transcendental Meditation to combat psychosocial stress, hypertension and cardiovascular disease.

Schneider RH, Norris KC, Brook RD

Nat Rev Cardiol · 2026 Jan · PMID 41266880 · Publisher ↗

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Mouse substrain is ceNNTral to the cardiac response to transverse aortic constriction.

Poss AM

Nat Rev Cardiol · 2026 Feb · PMID 41261121 · Publisher ↗

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Integration of cardiovascular and mental health: from concepts to the clinic.

Hahad O, Abohashem S

Nat Rev Cardiol · 2026 Jan · PMID 41225038 · Publisher ↗

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Gene-environment interactions in the pathogenesis of cardiovascular disease.

Ahmad S, Rukh G

Nat Rev Cardiol · 2026 Jan · PMID 41203938 · Publisher ↗

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Colchicine in patients with cardiovascular disease.

Muroke VJM

Nat Rev Cardiol · 2026 Jan · PMID 41188485 · Publisher ↗

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Safely implementing new knowledge from trials of withholding β-blockers after myocardial infarction.

Cleland JGF

Nat Rev Cardiol · 2025 Dec · PMID 41125934 · Publisher ↗

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Cardiovascular disease prevention in China: challenges and opportunities in the artificial intelligence-enabled digital health era.

Zhao D, Zhang Y, Wang J … +3 more , He L, He Y, Ma C

Nat Rev Cardiol · 2026 May · PMID 41094208 · Publisher ↗

Over the past decade, substantial progress has been made in China in improving health-care infrastructure and enacting national policies focused on the prevention of cardiovascular disease (CVD) and other non-communicabl... Over the past decade, substantial progress has been made in China in improving health-care infrastructure and enacting national policies focused on the prevention of cardiovascular disease (CVD) and other non-communicable chronic diseases. Nevertheless, a considerable gap remains between the national goals for cardiovascular health and the burden of CVD in the Chinese population. The rapid advances in digital health-care platforms and the increasing adoption of artificial intelligence (AI) technologies across various health-care sectors underscore the potential for these innovative approaches to address existing challenges in CVD prevention. In this Review, we summarize the major challenges in implementing preventative strategies and highlight the core obstacles to achieving the national goals for cardiovascular health in China. Furthermore, we discuss the potential value of, and the challenges associated with, digital health-care and AI technology in the implementation of preventative strategies in China and other countries with similar needs.

Resolutions from the Women As One RISE 2025 conference for women in cardiology.

Mehran R, Bowman K, Raona V … +2 more , Rubini Giménez M, Zühlke L

Nat Rev Cardiol · 2025 Dec · PMID 41087730 · Publisher ↗

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AQUATIC findings sink antiplatelet therapy for patients with chronic coronary syndrome requiring oral anticoagulants.

Parker WAE, Storey RF

Nat Rev Cardiol · 2025 Dec · PMID 41083609 · Publisher ↗

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LOX1 blockade does not modify atherosclerosis progression in patients with MI.

Fernández-Ruiz I

Nat Rev Cardiol · 2025 Dec · PMID 41073697 · Publisher ↗

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The global cardiovascular-liver-metabolic syndemic: epidemiology, trends and challenges.

Chew NWS, Mehta A, Goh R … +9 more , Koh J, Chen Y, Chong B, Chan MY, Khan MS, Muthiah MD, Butler J, Sanyal AJ, Sperling LS

Nat Rev Cardiol · 2026 Apr · PMID 41053364 · Publisher ↗

The growing epidemics of metabolic-dysfunction-associated steatotic liver disease, type 2 diabetes mellitus, obesity and cardiovascular disease are inextricably linked. Cardiovascular-liver-metabolic (CLM) diseases coexi... The growing epidemics of metabolic-dysfunction-associated steatotic liver disease, type 2 diabetes mellitus, obesity and cardiovascular disease are inextricably linked. Cardiovascular-liver-metabolic (CLM) diseases coexist and interact to constitute a synergy of epidemics (a syndemic), with shared mechanisms and socioeconomic influences. The goal of this Review is to construct this complex public-health issue into a unified framework, using epidemiological data to illustrate the current burden of disease and the trends in the CLM syndemic and to make projections for the future. We also discuss the challenges of promoting CLM health and the need for shared solutions. The proposed micro-meso-macro framework integrates strategies to improve risk prediction and precision prevention and to disentangle the competing risks within the CLM construct (micro), while keeping communities at the heart of CLM health promotion by ensuring access to healthy foods, healthy environments and metabolic interventions (meso). In addition, we propose interventions to eliminate inequities in the social and commercial determinants of health, from communities to healthcare systems (macro). Thus, multisystem interventions could address the trajectories of the CLM disease epidemics simultaneously.

Takotsubo syndrome: pathophysiological insights and innovations in patient care.

Omerovic E, Redfors B

Nat Rev Cardiol · 2026 Apr · PMID 41039160 · Publisher ↗

Takotsubo syndrome, also known as broken heart syndrome, was first described in Japan in the 1990s and presents as acute heart failure caused by transient contractile dysfunction that mimics acute coronary syndrome but i... Takotsubo syndrome, also known as broken heart syndrome, was first described in Japan in the 1990s and presents as acute heart failure caused by transient contractile dysfunction that mimics acute coronary syndrome but is not caused by acute coronary obstruction. Takotsubo syndrome accounts for as much as 2-3% of suspected coronary syndromes and is considerably more common in women than in men. In this Review, we discuss the historical background and clinical features of Takotsubo syndrome and our evolving understanding of its pathophysiology. The hallmark feature of Takotsubo syndrome is transient left ventricular dysfunction linked to emotional or physical stressors. Despite advances in our understanding of the clinical presentation and possible pathophysiological mechanisms of Takotsubo syndrome, this condition continues to challenge our fundamental understanding of human anatomy and physiology, particularly regarding how acute emotional and physical stressors can trigger such profound cardiovascular dysfunction. Although the definitive mechanisms remain elusive, current hypotheses largely centre on catecholamine surges and sympathetic nervous system hyperactivation. Diagnostic criteria have been developed by the Mayo Clinic and European Society of Cardiology working groups but, in the absence of randomized controlled clinical trials, no specific treatments for Takotsubo syndrome are available. In this Review, we highlight the complex pathophysiology of Takotsubo syndrome, with an emphasis on the interplay between emotional stress and cardiac health, and call for the development of evidence-based management protocols.

Multimodal, device-based therapeutic targeting of the cardiovascular autonomic nervous system.

Paton JFR, Żera T, Vadigepalli R … +2 more , Herring N, Paterson DJ

Nat Rev Cardiol · 2026 Apr · PMID 41034673 · Publisher ↗

The miniaturization of implantable sensors and actuators, combined with advances in interactive modelling and high-resolution imaging, is propelling the use of medical devices for counteracting impaired neural control of... The miniaturization of implantable sensors and actuators, combined with advances in interactive modelling and high-resolution imaging, is propelling the use of medical devices for counteracting impaired neural control of the cardiovascular system. In this Review, we discuss the current effectiveness of this technology for modulating autonomic activity in numerous cardiovascular conditions, including high blood pressure, heart failure and cardiac arrhythmias. We advocate for smarter closed-loop bionic devices fitted with feedback from multiple sensors to allow adaptive, state-dependent control, and discuss how the adoption of artificial intelligence technology would facilitate auto-personalization to meet the needs of patients. We also describe how transcriptomics of autonomic circuits can guide device-based approaches. Finally, the use of stem cell therapies to target sympathetic circuits more precisely will help to optimize the therapeutic effects of autonomic modulation for the treatment of arrhythmia. For bioelectronic medicine to achieve clinical utility in neurocardiology, these innovations must demonstrate improved efficacy beyond that offered by contemporary interventions.

Resistin-like molecule-γ promotes arrhythmia and cell death after MI.

Huynh K

Nat Rev Cardiol · 2025 Dec · PMID 40987867 · Publisher ↗

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