Tidsskr. Nor. Laegeforen. [JOURNAL]

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Norheim G, Grenersen MP

Tidsskr Nor Laegeforen · 2026 Jun · PMID 42343630 · Publisher ↗

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Correction: Management of acute epistaxis.

Lobekk OK, Lobekk HM, Aambakk J … +4 more , Fiskå RS, Steinbakk VH, Mjelle KES, Steinbakk AH

Tidsskr Nor Laegeforen · 2026 Jun · PMID 42343629 · Publisher ↗

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A woman in her 70s with chest pain and elevated troponin T levels.

Bertelsen BC, Qvigstad E, Larstorp ACK … +1 more , Ihle-Hansen H

Tidsskr Nor Laegeforen · 2026 Jun · PMID 42343616 · Publisher ↗

BACKGROUND: Troponin T is a biomarker of myocardial injury. In patients presenting with chest pain and elevated troponin T, acute coronary syndrome (ACS) is often suspected. However, alternative explanations must be cons... BACKGROUND: Troponin T is a biomarker of myocardial injury. In patients presenting with chest pain and elevated troponin T, acute coronary syndrome (ACS) is often suspected. However, alternative explanations must be considered. CASE PRESENTATION: A woman in her seventies was admitted to hospital with retrosternal chest pain and an initial elevated troponin T of 108 ng/L. Electrocardiogram and coronary angiography were normal. Further elevation to 263 ng/L raised suspicion of myopericarditis, even though cardiac MRI and echocardiography revealed no definitive pathology. Despite clinical improvement, troponin T remained persistently elevated at around 270 ng/L. Upon readmission 14 months later, troponin T levels were still elevated (89 ng/L) without corresponding symptoms. High-sensitivity troponin I was normal. Further biochemical testing revealed troponin T bound to IgG, consistent with macrotroponin and explaining the false elevation. INTERPRETATION: This case highlights the importance of considering biochemical factors when troponin results are inconsistent with clinical findings. Although macrotroponin was identified, the underlying cause of the initial elevated troponin T remains uncertain. The absence of cardiac findings suggests a non-cardiac origin, and the moderate, persistent elevation in creatine kinase raises the possibility of an underlying myopathic process. Diagnostic uncertainty can arise when biomarkers do not match the clinical picture.

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More systematic follow-up after childbirth.

Bentsen I, Waldum Å, Sugulle M … +2 more , Klovning A, Staff A

Tidsskr Nor Laegeforen · 2026 Jun · PMID 42343615 · Publisher ↗

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Askim K

Tidsskr Nor Laegeforen · 2026 Jun · PMID 42343614 · Publisher ↗

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Mousavi A

Tidsskr Nor Laegeforen · 2026 Jun · PMID 42343613 · Publisher ↗

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Tran E, Berger MF, Rygh E

Tidsskr Nor Laegeforen · 2026 Jun · PMID 42343612 · Publisher ↗

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Mestl H

Tidsskr Nor Laegeforen · 2026 Jun · PMID 42343611 · Publisher ↗

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Brochmann EH, Drivenes JL

Tidsskr Nor Laegeforen · 2026 Jun · PMID 42343610 · Publisher ↗

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Ductus arteriosus aneurysm in a neonate with inspiratory stridor.

Thayaparan S, Lamens SS, Pochylski FT … +2 more , Aukland SM, Ryste ST

Tidsskr Nor Laegeforen · 2026 Jun · PMID 42343609 · Publisher ↗

BACKGROUND: Inspiratory stridor in neonates is typically attributable to laryngomalacia, vocal cord paralysis, or subglottic stenosis. Less commonly, vascular anomalies can cause inspiratory stridor. CASE PRESENTATION: A... BACKGROUND: Inspiratory stridor in neonates is typically attributable to laryngomalacia, vocal cord paralysis, or subglottic stenosis. Less commonly, vascular anomalies can cause inspiratory stridor. CASE PRESENTATION: A neonate developed progressive, position-dependent inspiratory stridor and episodic oxygen desaturation on the third day of life. Laryngomalacia was excluded on initial investigation. Imaging revealed a ductus arteriosus aneurysm that was likely compressing the left recurrent laryngeal nerve, resulting in left-sided vocal cord paralysis. Despite marked respiratory symptoms, the neonate improved spontaneously and was managed conservatively. INTERPRETATION: This case underscores the importance of considering vascular anomalies - such as a ductus arteriosus aneurysm - in the differential diagnosis of neonatal stridor, particularly when symptoms are progressive or position-dependent. Timely recognition and targeted imaging facilitate accurate diagnosis, prevent unnecessary interventions and enable individualised management.

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